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Circulating and locally produced IGF-1 mediates many of GHs downstream effects and also feeds back negatively on both the hypothalamus and pituitary to restrain further GH release

Conclusions In summary, this study demonstrates the first evidence that exogenous GABA mitigates TS-like behavior in TS model rats by modulating harmful intestinal flora and striatal GABA-glutamate metabolism, central inflammatory state, and directly enhancing neuroprotective metabolites

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