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The strict relationship between CVS and pediatric primary headaches is supported by a study ( Comorbidities CVS is probably not the result of a single pathogenetic mechanism, rather the common final clinical picture (cyclical emesis attacks) of different physiopathological pathways, with different threshold, and many triggers can be able to elicit it (79)

The tested peptide led to a more than threefold decrease in the viability of senescent cells compared to proliferating cells, with an IC50 concentration (for senescent cells) of 8 M [128]